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중국의 동북공정에 관해 게시판에 글올립니다.~~

안녕하세요. 평소에 중국과 우리나라의 역사분쟁인 동북공정에 대해 관심이 많은 대학생 중의 한 사람입니다. 현제 베이징 올림픽이 얼마 남지 않은 시점에서 중국의 동북공정 사업이 잠잠하고 있지만 조만간 이 문제는 다시 붉어질거라고 생각합니다. 현재 중국의 동북공정 문제는 단순한 역사분쟁을 넘어서 여러 복합적인 사항을 가지고 있습니다. 동북공정을 통해 만약 북한의 붕괴같은 급변사태시 중국이 개입할 여지가 생기고 북한 핵문제 또한 거론시되고 있는 상황입니다. 더 나아가 역사문제 뿐만 아니라 고조선, 고구려의 역사가 중국사에 속해있다는 주장을 피면서 그 뿌리에서 갈라져 나온 백제, 신라의 역사까지 중국사에 포함시키려는 움직임을 보이면서 한반도의 민족성과 역사성을 중국에 포함시키려까지 하고 있습니다. 이러한 상황에서 ‘한사군’의 위치에 관한 쟁점이 동북공정에 핵심이 되고 있습니다. 한나라 무제가 고조선을 멸망시키고 설치했다는 4개의 군 중에 하나인 한사군의 위치가 현재 우리나라 평양에 위치했었다는 주장과 만리장성 끝 지역인 동쪽 산해관 지역에 위치 했었다는 주장이 첨애하게 대립하고 있는 실정입니다. 안타까운 점은 몇몇 우리나라 주류학계에서도 한사군이 평양에 위치했었다는 중국의 주장을 뒷받침해주고 있다는 사실에 놀랍기도 하고 많은 생각이 들기도 했습니다. 최근 미국산 소고기 문제 등 국내외로 쟁점하 되고 있는 사항들이 많지만 우리의 올바른 역사를 되찾는 것도 굉장히 중요하다고 생각합니다. 이러한 점에서 동북공정에 대한 의견과 대학생으로서 대처해야 할 점들은 어떤 것이 있는지 알려주셨으면 합니다.

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symptoms for shingles <a href=http://www.simplyfired.com/story/8668/>soma buy</a> Compound Total Amount (g) Iron Content Male (g) Iron Content Female (g) Percent of total iron Heme compound Hemoglobin Myoglobin (muscle hemoglobin)- Heme enzymes and cytochromes 800405.8 2.40.140.01 1.70.120.01 65-80%3.5%0.5% Nonheme Compounds Transferrin Iron �� sulfur proteins Ferritin 7.5-3.0 0.003-0.7- 15 0.003-0.7- 15 0.01%-- Hemosiderin - - - - Total storage iron - 1.0 �� 1.5 1.2. �� 1.5 30% Total iron 4.0 2.0 100% Iron stores :(Iron is stored in the form of ferritin in hepatocytes and hemosiderin in reticulo endothelial system)Newborn �� 80mg / kg6months to 2yerar �� Nil Childhood �� 5mg / kg 15 - 30 years �� 10 �� 12 mg / kg (Low in women) It takes 7 years or more for a man to deplete body iron stores solely due to lack of dietary intake.Cellular from homeostasis Low cellular iron levels�밒RP 1 binds to iron response element of mRNAs which code for transferrin receptor and ferritin.(When Fe is abundant IRP 1 contains iron sulphur cluster, and it acts as aconitase and has no affinity for iron �� response element of MRNA)�밫his bindingProtects transferrin receptor mRNA from cytoplasmic degradationInhibits translation of ferritin mRNA�밒ncreased iron uptake and storageOther mRNAs having iron responsive elements (IRE)쨌 Erythroid and ALA synthase mRNA 쨌 Mitochondrial aconitase mRNA (It interconverts citrate and isocitrate) 쨌 DMT 1 쨌 Ferroportin 1 MRNA Transferrin:It is 棺 globulin synthesized in liver Single chain polypeptide, present in plasma MW �� 79, 500 Concentration-1.8��2.6g/L Synthesized in liver ( synthesis in inversely related to iron stores)2 Ferric atoms can bind to each molecule Function �� Transport of iron Binding sites (C & N terminus) contain 3 tyrosine, 2 histidine & 1 arginine group.Binding of iron involves simultaneous binding of bicarbonate Plasma half life of transferrin in 5 days Each gram of transferrin can bind to 1.25 mg of iron. Transferrin receptor:M W �� 18500 Transmembrane protein with 2 subunits, each bind to 1 molecule of transferrinIdentified with anti C D 71 antibodies. Transferrin has anther receptor called TFR2, which acts as homeostatic iron sensor, regulating hepcidin synthesis in response to diferric transferrin concentration.Ferritin v It is a major storage form of iron found in bone marrow, spleen and liver v It is colorless and not visible microscopically v It gives a faint bluish tint to tissues, when stained with ferrocyanide method v It has outer coat of protein known as apoferritin and core contains iron. v Molecular weight is 450000 v Apoferritin contains 24 subunits (There are 2 immunologically distinct types of subunits �� H & L, which are coded by genes on chromosome 11 and 19 respectively) v Each molecule of ferritin contains up to 4500 iron molecules. v Iron in ferritin is present as hydrous ferric oxide phosphate v Internal cavity of ferritin molecule communicates with exterior via 6 channels, through which iron enters and leaves. v Various isoferritins can be separated by electrophoresis and difference in them is regarding content of H and L submits o Heart and Red cell ferritin are rich in H (Acidic) o Liver, spleen and serum ferritin are rich in L (Basic) o Mitochondrial ferritin (Gene on chromosome 5 is a novel H type ferritin ) v Entire ferritin molecule is degraded within lysosomes to release iron v Amount of circulating ferritin parallels concentration of storage iron in body. (1 ng / ml indicates 8mg of stored iron) v Less than 12ng / mg do not correlate with storage iron, because all stores are depleted at that level v Normal level �� 15-300 ng / ml Hemosiderin:v It is a water insoluble heterogeneous iron protein aggregate v 50% of its weight is composed of iron. v In unstained tissue specimens it appears as irregular golden brown granules and stains blue with Prussian blue iron stain. v With aging of ferritin, apoferritin denatures and hemosiderin is formed. Enzymes containing iron:v Cytochromes a, b, c v Succinate dehydrogenase v Cytochrome oxidase v Cytochrome P 450 -Hydroxylation & detoxification of drugs -Cyclooxygenase is involved inprostaglandin synthesis v Catalase v Lactoperoxidase v Trytophan pyrrolase- Oxidation of tryptophan to formylkynurenine v Iron sulphur proteins - xanthine oxidase -NADH2-Aconitasev Ribonucleotide reductase �� key enzyme in DNA Synthesis Due to decreased iron depended enzymes, there is Mitochondrial swelling in different cells Poor lymphocyte transformation & diminished cell mediated immunity.Impaired intracellular killing of bacteria by neutrophils Impaired mental development & function in infants.
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cozaar generic <a href=http://www.simplyfired.com/story/8657/>how is ultram better tham tramadol</a> In the Gemzar plus cisplatin versus etoposide plus cisplatin study, dose adjustments occurred with 20%혻of Gemzar injections and 16%혻of cisplatin injections in the Gemzar plus cisplatin arm compared with 20%혻of etoposide injections and 15%혻of cisplatin injections in the etoposide plus cisplatin arm. With a median of 5혻cycles of Gemzar plus cisplatin treatment, 15혻of 69 patients혻(22%) experienced 15혻hospitalizations due to possibly treatment�뱑elated adverse events. With a median of 4혻cycles of etoposide plus cisplatin treatment, 18혻of 66혻patients혻(27%) experienced 22혻hospitalizations due to possibly treatment�뱑elated adverse events. In patients who completed more than one cycle, dose adjustments were reported in 81%혻of the Gemzar plus cisplatin patients, compared with 68%혻on the etoposide plus cisplatin arm. Study discontinuations for possibly drug�뱑elated adverse events occurred in 14%혻of patients on the Gemzar plus cisplatin arm and in 8% of patients on the etoposide plus cisplatin arm. The incidence of myelosuppression was increased in frequency with Gemzar plus cisplatin treatment혻(~90%) compared to that with the Gemzar monotherapy (~60%). With combination therapy Gemzar dosage adjustments for hematologic toxicity were required more often while cisplatin dose adjustments were less frequently required.
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